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Von Hippel-Lindau (VHL)

Von Hippel-Lindau (VHL) syndrome, also known as Von Hippel-Lindau disease, is a rare genetic disorder that causes the development of multiple benign (non-cancerous) and malignant (cancerous) tumour throughout the body. It is caused by a mutation to the Von Hippel-Lindau gene, which is a tumour suppressor gene located on chromosome three.

Familial cancer syndromes, also known as hereditary cancer syndromes, are rare conditions that cause an increased risk of cancer as the result of inherited genetic mutations in certain cancer-related genes. They can affect both adults and children, however they generally develop in people at a younger age than normal. While familial cancer syndromes are not classified as cancer, they are equally as severe and can be life-threatening as they are associated with the development of various tumours throughout the body. Having a familial cancer syndrome does not guarantee the development of cancer, however the risk of developing cancer is higher than those who do not have a familial cancer syndrome.

VHL tends to affect the sexes equally, and is generally diagnosed around the age of 26. However, anyone can get this disease.

Types of Von Hippel-Lindau

VHL can be categorised into five subtypes, based on the likelihood of developing certain types of tumours.

Type 1A Von Hippel-Lindau

Type 1A VHL is characterised by low risk of developing pheochromocytomas, but higher risk for developing RCC, haemangioblastoma of the central nervous system (CNS), pancreatic cysts, and pNETs.

Type 1B Von Hippel-Lindau

Type 1B VHL is characterised by low risk of developing pheochromocytomas and RCCs, but higher risk for developing CNS haemangioblastomas, pancreatic cysts, and pNETS.

Type 2A Von Hippel-Lindau

Type 2A VHL is characterised by low risk of RCC, but high risk for pheochromocytomas. This may occur with or without the development of the other tumours listed above.

Type 2B Von Hippel-Lindau

Type 2B VHL is characterised by high risk for pheochromocytoma and RCC. This may occur with or without the development of the other tumours listed above.

Type 2C Von Hippel-Lindau

Type 2C VHL is characterised by high risk for pheochromocytoma, with or without the risk the risk of RCCs and haemangioblastomas.

VHL is most commonly associated with the development of haemangioblastomas, however it has also been linked to other types of tumours. Some examples include:

Treatment

When cancers are detected, they are staged and graded based on size, metastasis, and how the cancer cells look under the microscope. Staging and grading helps your doctors determine the best treatment for you. However, each patient with VHL will present with a unique disease behaviour, with varying tumour locations and symptoms. As such, there is no one treatment method that will work for everyone, and there is no standard staging system for this disease. Instead of staging and grading, your doctor will recommend a treatment plan based on the following factors:

  • Type of tumours present.
  • Whether the tumours are malignant (cancerous) or benign (non-cancerous).
  • Tumour location.
  • Whether or not malignant tumours have metastasised.
  • Your age.
  • General health.
  • Your treatment preferences.

Your doctor may also recommend genetic testing, which analyses your tumour DNA and can help determine which treatment has the greatest chance of success. They will then discuss the most appropriate treatment option for you.

Treatment options for tumours associated with VHL may include:

  • Surgery to remove as much of the tumour(s) as possible – this will vary based on tumour location.
  • Radiation therapy.
  • Chemotherapy.
  • Ablation therapy.
  • Clinical trials.
  • Palliative care.

Cancer Screening

Once a diagnosis of VHL has been confirmed, implementing a targeted screening plan becomes essential due to the increased risk of developing certain cancers.  The content of this plan will vary from person to person based on the genetic mutation involved, your family’s history of cancer and the types of cancers that may be present. It will also outline the routine tests you should have and how regularly you should have them.  Some recommendations for VHL may include:

  • Annual physical examination starting before the age of 10.
  • Annual neurological examination starting before the age of 10.
  • Annual blood pressure measurement starting before the age of 10.
  • MRI of the brain and spine every two years starting at age 11.
  • Annual eye exam starting at the age of one.
  • Abdominal MRI assessing the kidneys, pancreas and adrenal glands every two years starting at the age of 15.
  • Annual measurement of metanephrine levels (either via a 24-hour urine test or a blood test) starting at age five.
  • Hearing assessment every 2-3 years starting at age 11 – annually if symptoms of hearing loss, vertigo and/or tinnitus are present.
  • MRI of the internal auditory canals in asymptomatic people between the ages of 15-20 or as soon as symptoms of hearing loss, vertigo and/or tinnitus are present.

Screening options for VHL may evolve as new technologies are developed and our understanding of the condition grows. It is essential to discuss your individual circumstances with your healthcare team to determine the most appropriate screening plan for you.

Risk factors

VHL is caused by a genetic mutation in the VHL gene, which acts as a tumour suppressor in the body. It is an autosomal dominant disease, which means you have a 50% chance of developing the condition if one of your parents carries the mutation.

Symptoms

The symptoms of VHL often vary by the type(s) of tumours present. General symptoms of VHL may include:

  • Difficulties with balance and/or coordination.
  • Impaired vision.
  • Muscle weakness and/or pain in the affected area.
  • Headaches.
  • Back aches.
  • Neck pain.
  • Impaired bowel and/or bladder function.
  • Numbness and/or tingling in affected areas.
  • Haematuria.
  • Unexplained weight loss/loss of appetite.
  • Fatigue.
  • Fevers.
  • Hypertension.
  • Unexplainable sweatiness.
  • Tachycardia.
  • Hearing loss.
  • Tinnitus (ringing in the ears).
  • Vertigo (dizziness).

Symptoms related to specific tumours can be found on our knowledgebase.

Not everyone with the symptoms above will have cancer, but see your general practitioner (GP) if you are concerned.

Diagnosis

If your doctor suspects you have tumours associated with VHL, they may order some of the following tests to confirm the diagnosis and refer you to a specialist for treatment. The tests required for diagnosis will often vary based on the symptoms present, and where the tumour(s) are suspected to be located.

  • Physical examination.
  • Genetic testing.
  • Neurological examination.
  • Eye examination.
  • Endocrine studies.
  • Imaging tests, potentially including:
    • MRI (magnetic resonance imaging).
    • CT (computed tomography) scan.
    • PET (positron emission tomography) scan.
    • Ultrasound.
  • Exploratory procedures, such as an endoscopy.
  • Biopsy.

References

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