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Familial Adenomatous Polyposis (FAP)

Familial Adenomatous polyposis (FAP) is a rare genetic condition characterised by the development of hundreds to thousands of adenomatous colorectal polyps and a predisposition to malignancy (cancer), most notably colon cancer. It is caused by an alteration in the adenomatous polyposis coli (APC) gene, which is a type of tumour suppressor gene.

Familial cancer syndromes, also known as hereditary cancer syndromes, are rare conditions that cause an increased risk of cancer as the result of inherited genetic mutations in certain cancer-related genes. They can affect both adults and children, however they generally develop in people at a younger age than normal. While familial cancer syndromes are not classified as cancer, they are equally as severe and can be life-threatening as they are associated with the development of various tumours throughout the body. Having a familial cancer syndrome does not guarantee the development of cancer, however the risk of developing cancer is higher than those who do not have a familial cancer syndrome.

FAP is generally diagnosed equally among the sexes, and is often diagnosed around the age of 40. However, people with this condition may start developing colon polyps at approximately 16 years of age, and anyone can develop this disease.

Types of Familial Adenomatous Polyposis

There are several types of FAP, which are categorised by the number of colorectal polyps that develop and by any disease manifestations outside of the gastrointestinal tract.

FAP Subtypes

There are two distinct subtypes of FAP that present with polyps solely in the gastrointestinal tract.

Classic FAP

Classic FAP is the most common subtype of this disease, and is characterised by the development of over 100 colorectal polyps (benign tissue growths) that generally start developing around the age of 16. People with classic FAP may require an annual colonoscopy to screen for colorectal cancer.

Attenuated FAP (AFAP)

Attenuated FAP (AFAP) is a less common subtype of this disease, and is characterised by the development of less than 100 colorectal polyps (generally between 20-100). People with this subtype tend to develop polyps later in life, and the polyps are generally found in the proximal colon. While AFAP is considered a milder form of FAP, regular monitoring is still important to ensure the polyps don’t become cancerous.

FAP Variants

There are two distinct variants of FAP that also present with polyps in the gastrointestinal tract, however they also present with tumours in other areas of the body.

Gardner Syndrome

Gardner syndrome is a variant of familial adenomatous polyposis (FAP) that has the characteristics of FAP with additional tumours outside of the gastrointestinal tract, including osteomas (benign bone tumours), as well as various skin and soft tissue tumours. For more information on Gardner syndrome, please refer to the Rare Cancers Australia Gardner Syndrome page.

Turcot Syndrome

Turcot syndrome is a variant of familial adenomatous polyposis (FAP) that has the characteristics of FAP with an additional risk of developing brain tumours such as glioblastomas and medulloblastomas. For more information on Turcot syndrome, please refer to the Rare Cancers Australia Turcot Syndrome page.

FAP is most commonly associated with the development of colorectal cancers, however it has also been linked to other types of tumours. Some examples include:

Treatment

When cancers are detected, they are staged and graded based on size, metastasis, and how the cancer cells look under the microscope. Staging and grading helps your doctors determine the best treatment for you. However, each patient with FAP will present with a unique disease behaviour, with varying tumour locations and symptoms. As such, there is no one treatment method that will work for everyone, and there is no standard staging system for this disease. Instead of staging and grading, your doctor will recommend a treatment plan based on the following factors:

  • Type of tumours present.
  • Whether the tumours are malignant (cancerous) or benign (non-cancerous).
  • Tumour location.
  • Whether or not malignant tumours have metastasised.
  • Your age.
  • General health.
  • Your treatment preferences.

Your doctor may also recommend genetic testing, which analyses your tumour DNA and can help determine which treatment has the greatest chance of success. They will then discuss the most appropriate treatment option for you.

Treatment options for tumours associated with FAP may include:

  • Surgery to remove as much of the tumour(s) as possible – these will vary based on tumour type and location.
  • Chemotherapy.
  • Radiation therapy.
  • Hormone therapy.
  • Nonsteroidal anti-inflammatory drugs (NSAIDs).
  • Clinical trials.
  • Palliative care.

Cancer Screening

Once a diagnosis of FAP has been confirmed, implementing a targeted screening plan becomes essential due to the increased risk of developing certain cancers.  The content of this plan will vary from person to person based on the genetic mutation involved, your family’s history of cancer and the types of cancers that may be present. It will also outline the routine tests you should have and how regularly you should have them.  Some recommendations for FAP may include:

  • Annual physical examination.
  • Annual colonoscopy or flexible sigmoidoscopy starting at age 10-12.
  • Gastroscopy every four years starting around 20-25 or after colonic polyps have been detected.
  • Thyroid ultrasounds every 2-5 years starting in late adolescence.
  • Abdominal ultrasound every 3-6 months until age five in patients with a family history of hepatoblastoma.

Screening options for FAP may evolve as new technologies are developed and our understanding of the condition grows. It is essential to discuss your individual circumstances with your healthcare team to determine the most appropriate screening plan for you.

Risk factors

FAP is caused by a genetic mutation in the APC tumour suppressor gene, which acts to inhibit cell growth to prevent overproduction of cells and the development of tumours. It is an autosomal dominant disease, which means that you have a 50% chance of developing the condition if one of your parents carries the mutation.

Symptoms

The symptoms of FAP often vary by the type(s) of tumours present. General symptoms of FAP may include:

  • The presence of hundreds of polyps in the colon.
  • Abdominal pain and/or discomfort.
  • Changes in bowel movements, potentially including:
    • Diarrhoea.
    • Constipation.
    • Feeling of incomplete bowel movement.
    • Thin bowel stools.
    • Blood in stools.
  • Rectal bleeding.
  • Unexplained weight loss/loss of appetite.
  • Fatigue.
  • Anaemia.
  • Nausea and/or vomiting.

Symptoms related to specific tumours can be found on our knowledgebase.

Not everyone with the symptoms above will have cancer, but see your general practitioner (GP) if you are concerned.

Diagnosis

If your doctor suspects you have tumours associated with FAP, they may order some of the following tests to confirm the diagnosis and refer you to a specialist for treatment. The tests required for diagnosis will often vary based on the symptoms present, and where the tumour(s) are suspected to be located.

  • Physical examination.
  • Genetic testing.
  • Blood tests.
  • Imaging tests, potentially including:
    • Ultrasound.
    • MRI (magnetic resonance imaging).
    • CT (computed tomography) scan.
  • Exploratory procedures, such as:
    • Colonoscopy.
    • Flexible sigmoidoscopy.
    • Other types of endoscopy as indicated.
  • Biopsy.

References

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